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To address these limitations, we propose the use of an Actor-Network Theory (ANT) perspective

The GSHGPX4 antioxidant system in the mitochondria and cytoplasm is an important mechanism of cellular resistance to ferroptosis

Ethanol and TNBS Synergize Promoting Mucosal GSH Depletion Having shown that TNBS reductive metabolism induces a burst of ROS, including hydrogen peroxide, we next determined the levels of GSH and mucosal injury during the colitis induced by TNBS in rats

Fourth, the manifestation of MetS exacerbates immune-related neurotoxicity, and the interaction between increased atherogenicity and IRS biomarkers significantly predicts the severity of the condition [140]