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(2014) screened 6,700 compounds against mTOR and found that CHE had the most substantial killing effect on mTORC1-overactive cells, inducing autophagy by inhibiting mTORC1 and increasing AMPK (Kim et al., 2011), which forms autophagosomes and converts LC3I to LC3II (Tanida et al., 2008

For clinicians and patients navigating these results, the goal is calibrated, systematic interpretation that respects the limits of the evidence while taking meaningful exposures seriously.