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After the addition of 105 mg (0.908 mmol) of N -hydroxysuccinimide (Sigma-Aldrich, Burlington, Massachusetts, USA), 103.6 mg (0.502 mmol) of N,N -dicyclocarbodiimide (Sigma-Aldrich, Burlington, Massachusetts, USA), and 0.140 mL (1.04 mmol) of triethylamine (TEA

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BA physiology is diversely altered in Insr P1195L/ + /HFD mice P1195L/ + As fat-specific insulin receptor deficient mice (FIRKO mice) failed to exhibit glucose intolerance, we examined the contribution of insulin resistance in liver

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